文献引用
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1) A rationally designed CD19 monoclonal antibody-triptolide conjugate for the treatment of systemic lupus erythematosus.
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2) Macrophage P2Y6R activation aggravates psoriatic inflammation through IL-27-mediated Th1 responses.
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3) Molecular glue triggers degradation of PHGDH by enhancing the interaction between DDB1 and PHGDH.
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4) AZI2 mediates TBK1 activation at unresolved selective autophagy cargo receptor complexes with implications for CD8 T-cell infiltration in breast cancer.
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5) CENPN suppresses autophagy and increases paclitaxel resistance in nasopharyngeal carcinoma cells by inhibiting the CREB-VAMP8 signaling axis.
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6) The NLRX1-SLC39A7 complex orchestrates mitochondrial dynamics and mitophagy to rejuvenate intervertebral disc by modulating mitochondrial Zn2+ trafficking.
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7) Kinome-wide siRNA screen identifies a DCLK2-TBK1 oncogenic signaling axis in clear cell renal cell carcinoma.
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8) DDX21 mediates co-transcriptional RNA m6A modification to promote transcription termination and genome stability.
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9) Osteopontin deficiency promotes cartilaginous endplate degeneration by enhancing the NF-κB signaling to recruit macrophages and activate the NLRP3 inflammasome.
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10) DNMT aberration-incurred GPX4 suppression prompts osteoblast ferroptosis and osteoporosis.
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11) Miltefosine reinvigorates exhausted T cells by targeting their bioenergetic state.
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12) Interorgan communication in neurogenic heterotopic ossification: the role of brain-derived extracellular vesicles.
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13) Lumbar instability remodels cartilage endplate to induce intervertebral disc degeneration by recruiting osteoclasts via Hippo-CCL3 signaling.
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14) Targeting TM4SF1 promotes tumor senescence enhancing CD8+ T cell cytotoxic function in hepatocellular carcinoma.
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15) Multifunctional Nanodrug-Mediated Immunotherapy in Microsatellite Stable Colorectal Cancer via Promoting m6A Modification and M1-Like Tumor-Associated Macrophages Polarization.