文献引用
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1) TJP3 promotes T cell immunity escape and chemoresistance in breast cancer: a comprehensive analysis of anoikis-based prognosis prediction and drug sensitivity stratification.
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2) КОРРЕКЦИЯ КОГНИТИВНЫХ НАРУШЕНИЙ И СИГНАЛЬНЫХ ПУТЕЙ В ГИППОКАМПЕ КРЫС С САХАРНЫМ ДИАБЕТОМ 2-ГО ТИПА С ПОМОЩЬЮ ИНТРАНАЗАЛЬНО ВВОДИМЫХ ИНСУЛИНА И ГАНГЛИОЗИДОВ.
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3) Exploring Senescence-Related Transcriptional Signatures to Uncover Molecular Subtypes and Key Genes in Hepatocellular Carcinoma.
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4) LncRNA AP000695.2 promotes glycolysis of lung adenocarcinoma via the miR-335-3p/TEAD1 axis: The role of AP000695.2 in glycolysis of lung adenocarcinoma.
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5) The antioxidant Glycitin protects against intervertebral disc degeneration through antagonizing inflammation and oxidative stress in nucleus pulposus cells.
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6) Restraint stress promotes nonalcoholic steatohepatitis by regulating the farnesoid X receptor/NLRP3 signaling pathway: Restraint stress promotes nonalcoholic steatohepatitis.
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7) 四川黑茶药膳配方通过重塑肠道菌群和短链脂肪酸代谢改善肥胖小鼠肾脏脂质紊乱.
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8) Intranasal Insulin and Gangliosides Correct Cognitive Impairments and Signaling Pathways in the Hippocampus of Rats with Type 2 Diabetes Mellitus.
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9) SNORA38b promotes proliferation, migration, invasion and epithelial-mesenchymal transition of gallbladder cancer cells via activating TGF-β/Smad2/3 signaling.
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10) [Sichuan Dark Tea-Based Medicated Dietary Formula Improves Obesity-Induced Renal Lipid Metabolism Disorder in Mice by Remodeling Gut Microbiota and Short-Chain Fatty Acid Metabolism].
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11) Eliminating the invading extracellular and intracellular FnBp+ bacteria from respiratory epithelial cells by autophagy mediated through FnBp-Fn-Integrin α5β1 axis.
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12) A pan-cancer analysis of RGR opsin expression and its downregulation associated with poor prognosis in glioma.
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13) 前列腺素D2对山羊黄体细胞内分泌功能及其凋亡相关基因表达的影响.
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14) Evaluation of the State of the Blood-Retinal Barrier during the Development of Signs of Age-Related Macular Degeneration in OXYS Rats.
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15) Blocking the CXCL1-CXCR2 axis enhances the effects of doxorubicin in HCC by remodeling the tumor microenvironment via the NF-κB/IL-1?/CXCL1 signaling pathway.