文献引用
-
1) Fenofibrate anti-RARα/RXRα dimerization to attenuate all-trans retinoic acid-induced hyperlipidemia and hepatic steatosis in mice.
-
2) Circular RNA circFNDC3B Modulates M2 Macrophage Activation and Intestinal Inflammation in Crohn's Disease.
-
3) Molecular mechanism underlying the specific RNA recognition of mitochondrial helicase DDX28 and its critical role in mitoribosomal biogenesis.
-
4) Exploring the role and potential mechanisms of SIK1 in cartilage damage in osteoarthritis based on metabolomics.
-
5) Recombinant Thrombomodulin Domain 1 Promotes Diabetic Corneal Wound Healing by Inhibiting HMGB1 Production and NLRP3 Inflammasome.
-
6) Assessing effects of microwave electromagnetic field in APP/PS1 mice by in vivo GABA-weighted imaging via variable delay multipulse-chemical exchange saturation transfer MRI.
-
7) AICAR improves depression-like behaviors and is associated with hippocampal AMPK activation and modulation of neurogenesis and neuroinflammation in a microbiota disruption model.
-
8) Antioxidant and anti-aging activities of Salvia miltiorrhiza Bunge callus extracts in H2O2-induced premature senescence and chronological aging models of human dermal fibroblasts.
-
9) CACNB2 overexpression suppresses atrial fibroblast activation and atrial fibrosis to mitigate atrial fibrillation.
-
10) Augmenting the Endogenous RBM3 Response Protects Against Burn-Induced Acute Kidney Injury by Enhancing Macrophage-Mediated Resolution of Inflammation.
-
11) USP14 and ELAVL1-induced stabilization of VDAC1 aggravates myocardial cell injury in diabetic cardiomyopathy.
-
12) The mechanosensitive ion channel Piezo1 promotes colitis by modulating oxidative stress and ferroptosis-related markers.
-
13) Transcription Factor GATA3 Ameliorates Sepsis-Associated Acute Respiratory Distress Syndrome by Activating Smad1/5-YAP Pathway via BMP9.
-
14) Dapagliflozin Attenuates Atherosclerosis Under Chronic Stress by Maintaining AKT/FoxO1 Pathway Through Downregulation of REDD1.
-
15) Combatting Silicosis Fibrosis: Methyl Gallate Suppresses Pathogenic Fibroblasts Through hnRNPA2/B1-MDM4-P53 Network Disruption.