文献引用
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1) Mitochondrial metabolism blockade nanoadjuvant reversed immune-resistance microenvironment to sensitize albumin-bound paclitaxel-based chemo-immunotherapy.
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2) The role of the microbiota–gut–brain axis in methamphetamine-induced neurotoxicity: Disruption of microbial composition and short-chain fatty acid metabolism.
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3) A rationally designed CD19 monoclonal antibody-triptolide conjugate for the treatment of systemic lupus erythematosus.
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4) Macrophage P2Y6R activation aggravates psoriatic inflammation through IL-27-mediated Th1 responses.
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5) Molecular glue triggers degradation of PHGDH by enhancing the interaction between DDB1 and PHGDH.
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6) AZI2 mediates TBK1 activation at unresolved selective autophagy cargo receptor complexes with implications for CD8 T-cell infiltration in breast cancer.
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7) CENPN suppresses autophagy and increases paclitaxel resistance in nasopharyngeal carcinoma cells by inhibiting the CREB-VAMP8 signaling axis.
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8) The NLRX1-SLC39A7 complex orchestrates mitochondrial dynamics and mitophagy to rejuvenate intervertebral disc by modulating mitochondrial Zn2+ trafficking.
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9) Kinome-wide siRNA screen identifies a DCLK2-TBK1 oncogenic signaling axis in clear cell renal cell carcinoma.
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10) DDX21 mediates co-transcriptional RNA m6A modification to promote transcription termination and genome stability.
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11) Osteopontin deficiency promotes cartilaginous endplate degeneration by enhancing the NF-κB signaling to recruit macrophages and activate the NLRP3 inflammasome.
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12) DNMT aberration-incurred GPX4 suppression prompts osteoblast ferroptosis and osteoporosis.
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13) Miltefosine reinvigorates exhausted T cells by targeting their bioenergetic state.
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14) Interorgan communication in neurogenic heterotopic ossification: the role of brain-derived extracellular vesicles.
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15) Lumbar instability remodels cartilage endplate to induce intervertebral disc degeneration by recruiting osteoclasts via Hippo-CCL3 signaling.