文献引用
-
1) Mitochondrial transfer of drug-loaded artificial mitochondria for enhanced anti-Glioma therapy through synergistic apoptosis/ferroptosis/immunogenic cell death.
-
2) Autophagy-induced NR2F1 activation promotes the apoptosis of lens epithelial cells and facilitates cataract-associated fibrosis through targeting STAT3.
-
3) Engineered probiotics remodel the intestinal epithelial barrier and enhance bacteriotherapy for inflammatory bowel diseases.
-
4) Selenium nanozyme-crosslinked composite hydrogel for promoting cartilage regeneration in osteoarthritis via an integrated 'outside-in' and 'inside-out' strategy.
-
5) Nanozyme cascade reaction system for diabetic retinopathy therapy via glycometabolism and microenvironment regulation.
-
6) Multifunctional Co-FeS2 nanozyme-hydrogel composite promotes diabetic wound healing by synergistic antioxidant, antibacterial, and anti-inflammation therapy.
-
7) Electrowritten topographical conduit integrated with 4-octyl itaconate for peripheral nerve immunomodulation and regeneration.
-
8) Injectable, antifouling granular zwitterionic hydrogels for preventing postoperative intrauterine adhesion and restoring fertility.
-
9) Dual-mechanism mucoadhesive hydrogel integrating pH-independent barriers and autophagy-driven immunotherapy for the treatment of ulcerative colitis.
-
10) IDO1 modulates pain sensitivity and comorbid anxiety in chronic migraine through microglial activation and synaptic pruning.
-
11) Roxadustat improves diabetic myocardial injury by upregulating HIF-1α/UCP2 against oxidative stress.
-
12) PARP9 exacerbates apoptosis and neuroinflammation via the PI3K pathway in the thalamus and hippocampus and cognitive decline after cortical infarction.
-
13) Microglial double stranded DNA accumulation induced by DNase II deficiency drives neuroinflammation and neurodegeneration.
-
14) THBS1 in macrophage-derived exosomes exacerbates cerebral ischemia-reperfusion injury by inducing ferroptosis in endothelial cells.
-
15) IFN-γ signaling links ventriculomegaly to choroid plexus and ependyma dysfunction following maternal immune activation.